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Dott.
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Corso biennale di
parodontologia ed implantologia
Scuola ARDEC
Rimini
Cristiano Tomasi DDS MSC Spec
Perio
Department of Periodontology
Insitute of Odontology
The Sahlgrenska Academy at Göteborg University
Box 450
SE 405 30 Göteborg,
Sweden
Phone +46.31.7733124
Fax +46.31.7733791
cristiano.tomasi@odontologi.gu.se
Dott. Cristiano Tomasi MSC Spec Perio
Studio Associato Tomasi e Bottamedi
Via Degli Orti, 15
I-38100 Trento
Italia
Tel +39.0461.239968
Fax +39.0461.268504
cris@cristianotomasi.it
Dott. Tomasi Cristiano, laureato con Lode in odontoiatria e p.d. nel 1991 presso l’Universitŕ di Verona. Ha conseguito il diploma program in Parodontologia ed in Implantologia presso la scuola Ariminum di Rimini nel 1996.
Ha ricevuto il diploma di specializzazione in Parodontologia clinica nel 2002 presso l’Universitŕ di Göteborg, Svezia.
Ha conseguito il titolo di Master of Science nel 2003 presso la stessa Universitŕ con una tesi sull’uso degli antibiotici locali ed un progetto di ricerca clinica sull’argomento.
Nel 2005 ha vinto il primo premio europeo EFP per la ricerca parodontale
riservato a specialisti presso scuole riconosciute dalla EFP (European
Federation of Periodontology) con l’articolo “Locally delivered doxycycline
improves the healing following non-surgical periodontal therapy in smokers”
pubblicato nel 2004 sul “Journal of Clinical Periodontology”.
Attualmente studente ricercatore per un PhD presso l’Universitŕ di Göteborg
con tutore Prof. Jan Wennström.
Ha pubblicato alcuni articoli ad argomento parodontale su riviste
internazionali peer reviewed. Partecipa ad alcuni progetti di ricerca
clinica sulla terapia implantare. Relatore presso il corso post graduate in
parodontologia clinica dell’Universitŕ di Goteborg. Relatore in
parodontologia ed implantologia presso il centro Ardec. Relatore in
congressi internazionali di parodontologia. Relatore presso il master in
implantologia dell’Universitŕ di Padova. Socio attivo della SIO. Socio della
SidP.
Dal 1992 lavora nel proprio studio odontoiatrico in Trento. Dal 1999 si occupa esclusivamente di parodontologia, piccola chirurgia orale e terapia implantare.
Consulente come parodontologo e per l’implantologia in altri studi privati.
Abstract:
AIM: To evaluate the incidence of disease
recurrence following a full-mouth pocket/root debridement approach with
ultrasonic instrumentation versus that following a traditional approach of
quadrant-wise scaling and root planing (Q-SRP) performed with hand
instrumentation.
METHODS: Thirty-seven patients were re-examined
1 year after the completion of a 6-month clinical trial comparing two
different treatment protocols: a 1-h session of full-mouth ultrasonic
debridement (UD--19 patients) or four sessions of Q-SRP with hand
instruments (Q-SRP--18 patients). At 3 months, re-instrumentation was
performed of pockets showing a remaining probing pocket depth (PPD) of > or
=5 mm using the same type of instruments as used during the initial
treatment phase. The clinical examinations comprised assessments of plaque,
bleeding on probing (BoP) and PPD.
The primary outcome variable was the incidence of recurrent diseased sites (i.e.,
sites showing PPD > or =5 mm and BoP+) between the post-treatment and 1-year
follow-up examinations. All sites that were healed (PPD < or =4 mm and BoP(-))
at the post-treatment examination were included in the study sample, with a
mean number of sites per patient of 23.5.
RESULTS: In the UD group, 29 (7%) out of 430
initially healed sites showed disease recurrence at the 1-year follow-up
examination compared with 47 (11%) of 440 sites in the Q-SRP group (p>0.05).
Twelve patients (63%) in the UD group presented recurrent diseased pockets,
compared with 14 patients (78%) in the Q-SRP group. Two or more recurrent,
diseased pockets were observed in nine patients in the UD group versus 11 in
the Q-SRP group. All but one of the smokers belonged to the group of
patients presenting recurrences. A tendency towards a higher mean plaque
score was observed for the patients with recurrent sites.
CONCLUSION: The study revealed no significant
difference in the incidence of recurrence of diseased periodontal pockets
between the full-mouth UD approach and the traditional approach of Q-SRP.
Abstract:
The main objective of the treatment of patients with periodontitis is to establish adequate infection control in the dentogingival area. Pocket/root instrumentation (scaling and root planing), combined with effective self-performed supragingival plaque control measures, constitute the basic treatment modalities, but also locally applied antiseptics and antibiotics may be utilized. The purpose of this article is to give an overview of current clinical concepts on
periodontal infection control.
Abstract:
BACKGROUND: The erbium-doped:yttrium, aluminum,
and garnet (Er:YAG) laser is considered a useful tool for subgingival
debridement because the laser treatment creates minimal damage to the root
surface and has potential antimicrobial effects. The aim of this randomized
controlled clinical trial was to evaluate clinical and microbiologic effects
of pocket debridement using an Er:YAG laser in patients during periodontal
maintenance.
METHODS: Twenty patients at a recall visit for maintenance were
consecutively recruited if presenting at least four teeth with residual
probing depth (PD) > or = 5 mm. Two pockets in each of two jaw quadrants
were randomly assigned to subgingival debridement using 1) an Er:YAG laser
(test) or 2) an ultrasonic scaler (control). The laser beam was set at 160
mJ with a pulse frequency of 10 Hz. Clinical variables were recorded at
baseline, 1 month, and 4 months after treatment. Primary clinical outcome
variables were changes in PD and clinical attachment level (CAL).
Microbiologic analysis of subgingival samples was performed at baseline, 2
days, and 30 days after treatment using a checkerboard DNA-DNA hybridization
technique against 12 periodontal disease-associated species.
RESULTS: The mean initial PD was 6.0 mm (SD:
1.2) in the test group and 5.8 mm (SD: 0.9) in the control group. At 1 month
post-treatment, the PD reduction was significantly greater for test than
control sites (0.9 versus 0.5 mm; P <0.05). The CAL gain also was
significantly greater (0.5 versus 0.06 mm; P <0.01). At the 4-month
examination, no significant differences were detected in PD reduction (1.1
versus 1.0 mm) or CAL gain (0.6 versus 0.4 mm). Both treatments resulted in
reduction of the subgingival microflora. No significant differences in
microbiologic composition were identified between the treatment groups at
various time intervals. Degree of treatment discomfort scored significantly
lower for the test than the control treatment modality.
CONCLUSION: The results of the trial failed to
demonstrate any apparent advantage of using an Er:YAG laser for subgingival
debridement, except less treatment discomfort perceived by the patients.
Abstract:
AIM: To evaluate the clinical efficacy of (i) a
single session of "full-mouth ultrasonic debridement" (Fm-UD) as an initial
periodontal treatment approach and (ii) re-instrumentation of periodontal
pockets not properly responding to initial subgingival instrumentation.
METHODS: Forty-one patients, having on the average 35 periodontal
sites with probing pocket depth (PPD) > or =5 mm, were randomly assigned to
two different treatment protocols following stratification for smoking: a
single session of full-mouth subgingival instrumentation using a
piezoceramic ultrasonic device (EMS PiezonMaster 400, A+PerioSlim tips) with
water coolant (Fm-UD) or quadrant scaling/root planing (Q-SRP) with hand
instruments . At 3 months, all sites with remaining PPD> or =5 mm were
subjected to repeated debridement with either the ultrasonic device or hand
instruments.
Plaque, PPD, relative attachment level (RAL) and bleeding following pocket
probing (BoP) were assessed at baseline, 3 and 6 months. Primary efficacy
variables were percentage of "closed pockets" (PPD< or =4 mm), and changes
in BoP, PPD and RAL.
RESULTS: The percentage of "closed pockets" was
58% at 3 months for the Fm-UD approach and 66% for the Q-SRP approach
(p>0.05). Both treatment groups showed a mean reduction in PPD of 1.8 mm,
while the mean RAL gain amounted to 1.3 mm for Fm-UD and 1.2 mm for Q-SRP
(p>0.05). The re-treatment at 3 months resulted in a further mean PPD
reduction of 0.4 mm and RAL gain of 0.3 mm at 6 months, independent of the
use of ultrasonic or hand instruments. The efficiency of the initial
treatment phase (time used for instrumentation/number of pockets closed) was
significantly higher for the Fm-UD than the Q-SRP approach: 3.3 versus 8.8
min. per closed pocket (p<0.01). The efficiency of the re-treatment session
at 3 months was 11.5 min. for ultrasonic and 12.6 min. for hand
instrumentation (p>0.05).
CONCLUSION: The results demonstrated that a
single session of Fm-UD is a justified initial treatment approach that
offers tangible benefits for the chronic periodontitis patient.
Abstract:
OBJECTIVE: The outcome of non-surgical
periodontal therapy is known to be inferior in smokers compared to
non-smokers. In the present study, the question was asked whether such a
difference in healing response may be less evident following adjunctive use
of locally delivered controlled-release doxycycline.
METHODS: One hundred and three patients (42
smokers, 61 non-smokers), each having at least eight periodontal sites with
PPD (probing pocket depth) > or =5 mm, were following stratification for
smoking randomly assigned to two different treatment protocols; non-surgical
scaling/root planing (Control) or ultrasonic instrumentation+application of
a 8.5% w/w doxycycline gel (Atridox trade mark ) (Test). Instructions in
oral hygiene were given to all patients. Clinical examinations of plaque,
PPD, clinical attachment level (CAL) and bleeding following pocket probing
were performed at baseline and after 3 months. Primary efficacy endpoints
were changes in PPD and CAL. Patient mean values were calculated as basis
for statistical analysis (multiple regression analyses).
RESULTS: The baseline examination revealed no
significant difference in mean PPD between treatment groups or between
smokers and non-smokers (mean PPD 5.7-5.9 mm). The mean PPD reduction in the
control group at 3-month was 1.1 mm (SD=0.45) for smokers and 1.5 mm (0.67)
for non-smokers. In the test group the PPD reduction was 1.4 mm (0.60) and
1.6 mm (0.45) for smokers and non-smokers, respectively. The mean CAL gain
for smokers and non-smokers amounted to 0.5 mm (0.56) and 0.8 mm (0.71),
respectively, in the control group, and to 0.8 mm (0.72) and 0.9 mm (0.82),
respectively, in the test group. Multiple regression analysis revealed that
smoking and initial PPD negatively influenced the treatment outcome in terms
of PPD reduction and CAL gain, while the use of doxycycline had a
significant positive effect.
CONCLUSION: Locally applied controlled-release
doxycycline gel may partly counteract the negative effect of smoking on
periodontal healing following non-surgical therapy.